What happens to your heart rate after you exercise and why?

When we workout, we typically think about muscles in our arms, legs, back or abdomen. However, these aren’t the only muscles that get stronger with regular exercise.

Your heart, which is a muscular organ, gets a workout during exercise, too. Your heart’s full-time job is to distribute blood throughout your entire body. When it beats harder and faster, it can grow stronger and more resilient.

When you’re stationary, your heart should beat between 50 to 100 times per minute to fall within the normal range. To narrow this down a little, most people’s hearts beat between 60 to 80 times per minute.

When you start your exercise, your muscles will start to work harder and demand more oxygen. This demand will cause sympathetic nerves to stimulate the heart to beat faster and with more force to increase overall blood flow. The sympathetic nerves will also stimulate the veins, causing them to compress. These narrower veins will increase the velocity at which blood flows through your body. This will ultimately increase the amount of blood returning to the heart.

How fast your heart pumps during exercise depends on numerous factors such as:

  • Your age
  • Your fitness level
  • Intensity of the workout
  • Your heart’s overall strength

Cardiac output is the rate of blood pumped by the heart. You can calculate this output by multiplying the rate at which the heart beats (heart rate) by the volume of blood the heart ejects with each beat (stroke volume). When your heart is pumping at full force, the cardiac output is about 20-25 liters per minute —compared to 5 liters a minute for a heart at rest.

Once you are done with your exercise, your heart rate will begin to slow to a normal pace.

Since your heart is a muscle, it can benefit from being “strained” to a certain degree which trains and prepares it to work harder when you need it to, but it also helps it to perform better – even while at rest. Frequent exercise will cause your heart to grow stronger, making it easier for it to distribute blood throughout your body. A stronger heart means that each time your heart beats, it pumps more and more blood throughout you body. This allows your heart to beat less, while still distributing the same amount of blood. All of this can keep your blood pressure under control.

Keep your heart healthy with regular check-ups by your doctor. Don’t have a doctor? Find one here.

What happens to your heart rate after you exercise and why?

A stress test should include measuring how quickly your elevated heart rate returns to normal after exercise.

Image Credit: Fuse/Fuse/Getty Images

Your heart rate slows down when you stop exercising. Your pulse two minutes after exercise is what is called your "recovery heart rate," and that rate will go down as you become more fit. The actual numbers vary because everyone's heart rate varies--even between similar people at similar fitness levels. In some people, the heart rate remains elevated after exercise, and electrical pulses from the heart are irregular. This is often a sign of potential health problems, according to the Harvard Medical School Family Health Guide.

Recovery Time

The heart rate of a typical individual will return to resting levels shortly after light or moderate exercise.The heart rates of athletes--particularly endurance-trained and strength-trained athletes--will return to normal after exercise faster than nonathletes.

Slow Recovery

A healthy person should see his heart rate decline by 15 to 20 beats per minute in the first minute after stopping exercise. Your "recovery heart rate"--the pulse you register two minutes after you stop exercising--is a measure of how fit you are. Say you run for 30 minutes at an average heart rate of 155 beats per minute and your heart rate two minutes after finishing is 95 beats per minute. As you become more fit, your heart rate will decrease faster after that 30-minute run, and your recovery heart rate will be lower at the two-minute mark. In healthy individuals, an effective program and healthy diet can also lower significantly resting heart rate.

Stress Test

Doctors use stress tests to examine how well a heart functions during exercise. A stress test uses a blood pressure cuff and an electrocardiograph to measure how exercise on a treadmill or exercise bike affects your blood pressure, heart rate and the heart's electrical activity. The test can reveal conditions--such as clogged arteries--that don't show up when the person's heart rate is normal. According to the Journal of Exercise Physiology, a slow return to a normal heart rate could mean the person has a great risk of developing diabetes, abdominal obesity, high cholesterol and other cardiovascular risks.

Abnormal Pulse

In recent years, doctors have started examining what happens to a patient's heart as it returns to its normal pulse rate after a stress test. While uncommon rhythms during exercise don't seem to be cause for concern, abnormal pulses in the heart ventricles that occur only after exercise can indicate a higher risk of imminent death, the Family Health Guide says.

Death Studies

In one study, 11 percent of the people who had this problem died within five years while only 5 percent of those who didn't show this characteristic died. In another study, those whose heart rates took longer than normal to recover were four times more likely to have died over the next three years, the Family Medical Guide reported.

Treatment

If your doctor notices an irregular rhythm during the 5- to 10-minute cool-down period after a stress test, she may take more proactive actions to control the factors that can trigger heart attack or stroke in the patient, such as high blood pressure, high cholesterol, high blood sugar, obesity and smoking. Your doctor may also order an echocardiogram to more closely examine your heart in action.

Exercise Helps

People who show signs of future heart problems may still be advised by their doctor to exercise. According to the University of New Mexico, exercise helps control and prevent high blood pressure, diabetes and other health problems. But most doctors will want to confer with their patients before a patient with potential heart problems begins or continues an exercise program.

1. Akselrod S, Gordon D, Ubel FA, Shannon DC, Berger AC, Cohen RJ. Power spectrum analysis of heart rate fluctuation: a quantitative probe of beat-to-beat cardiovascular control. Science 213: 220–222, 1981. doi: 10.1126/science.6166045. [PubMed] [CrossRef] [Google Scholar]

2. Ayada K, Watanabe M, Endo Y. Elevation of histidine decarboxylase activity in skeletal muscles and stomach in mice by stress and exercise. Am J Physiol Regul Integr Comp Physiol 279: R2042–R2047, 2000. [PubMed] [Google Scholar]

3. Barrett-O’Keefe Z, Kaplon RE, Halliwill JR. Sustained postexercise vasodilatation and histamine receptor activation following small muscle-mass exercise in humans. Exp Physiol 98: 268–277, 2013. doi: 10.1113/expphysiol.2012.066605. [PubMed] [CrossRef] [Google Scholar]

4. Bellenger CR, Fuller JT, Thomson RL, Davison K, Robertson EY, Buckley JD. Monitoring athletic training status through autonomic heart rate regulation: a systematic review and meta-analysis. Sports Med 46: 1461–1486, 2016. doi: 10.1007/s40279-016-0484-2. [PubMed] [CrossRef] [Google Scholar]

5. Billman GE. Heart rate variability - a historical perspective. Front Physiol 2: 86, 2011. doi: 10.3389/fphys.2011.00086. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

6. Billman GE, Huikuri HV, Sacha J, Trimmel K. An introduction to heart rate variability: methodological considerations and clinical applications. Front Physiol 6: 55, 2015. doi: 10.3389/fphys.2015.00055. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

7. Bosquet L, Merkari S, Arvisais D, Aubert AE. Is heart rate a convenient tool to monitor over-reaching? A systematic review of the literature. Br J Sports Med 42: 709–714, 2008. doi: 10.1136/bjsm.2007.042200. [PubMed] [CrossRef] [Google Scholar]

8. Brito LC, Queiroz AC, Forjaz CL. Influence of population and exercise protocol characteristics on hemodynamic determinants of post-aerobic exercise hypotension. Braz J Med Biol Res 47: 626–636, 2014. doi: 10.1590/1414-431X20143832. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

9. Brown SP, Clemons JM, He Q, Liu S. Effects of resistance exercise and cycling on recovery blood pressure. J Sports Sci 12: 463–468, 1994. doi: 10.1080/02640419408732196. [PubMed] [CrossRef] [Google Scholar]

10. Buck TM, Romero SA, Ely MR, Sieck DC, Abdala PM, Halliwill JR. Neurovascular control following small muscle-mass exercise in humans. Physiol Rep 3: e12289–e12289, 2015. doi: 10.14814/phy2.12289. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

11. Casonatto J, Goessler KF, Cornelissen VA, Cardoso JR, Polito MD. The blood pressure-lowering effect of a single bout of resistance exercise: a systematic review and meta-analysis of randomised controlled trials. Eur J Prev Cardiol 23: 1700–1714, 2016. doi: 10.1177/2047487316664147. [PubMed] [CrossRef] [Google Scholar]

12. Chan-Dewar F, Oxborough D, Shave R, Gregson W, Whyte G, Noakes T, George K. Evidence of increased electro-mechanical delay in the left and right ventricle after prolonged exercise. Eur J Appl Physiol 108: 581–587, 2010. doi: 10.1007/s00421-009-1264-6. [PubMed] [CrossRef] [Google Scholar]

13. Chen CY, Bonham AC. Postexercise hypotension: central mechanisms. Exerc Sport Sci Rev 38: 122–127, 2010. doi: 10.1097/JES.0b013e3181e372b5. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

14. Cheuvront SN, Kenefick RW. Dehydration: physiology, assessment, and performance effects. Compr Physiol 4: 257–285, 2014. doi: 10.1002/cphy.c130017. [PubMed] [CrossRef] [Google Scholar]

15. Coats AJ, Conway J, Isea JE, Pannarale G, Sleight P, Somers VK. Systemic and forearm vascular resistance changes after upright bicycle exercise in man. J Physiol 413: 289–298, 1989. doi: 10.1113/jphysiol.1989.sp017654. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

16. Compton D, Hill PM, Sinclair JD. Weight-lifters’ blackout. Lancet 2: 1234–1237, 1973. doi: 10.1016/S0140-6736(73)90974-4. [PubMed] [CrossRef] [Google Scholar]

17. Ely MR, Romero SA, Sieck DC, Mangum JE, Luttrell MJ, Halliwill JR. A single dose of histamine-receptor antagonists prior to downhill running alters markers of muscle damage and delayed onset muscle soreness. J Appl Physiol (1985) 122: 631–641, 2017. doi: 10.1152/japplphysiol.00518.2016. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

18. Emhoff CA, Barrett-O’Keefe Z, Padgett RC, Hawn JA, Halliwill JR. Histamine-receptor blockade reduces blood flow but not muscle glucose uptake during postexercise recovery in humans. Exp Physiol 96: 664–673, 2011. doi: 10.1113/expphysiol.2010.056150. [PubMed] [CrossRef] [Google Scholar]

19. Endo Y, Tabata T, Kuroda H, Tadano T, Matsushima K, Watanabe M. Induction of histidine decarboxylase in skeletal muscle in mice by electrical stimulation, prolonged walking and interleukin-1. J Physiol 509: 587–598, 1998. doi: 10.1111/j.1469-7793.1998.587bn.x. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

20. Fisher MM. The effect of resistance exercise on recovery blood pressure in normotensive and borderline hypertensive women. J Strength Cond Res 15: 210–216, 2001. [PubMed] [Google Scholar]

21. Focht BC, Koltyn KF. Influence of resistance exercise of different intensities on state anxiety and blood pressure. Med Sci Sports Exerc 31: 456–463, 1999. doi: 10.1097/00005768-199903000-00016. [PubMed] [CrossRef] [Google Scholar]

22. Fu Q, Arbab-Zadeh A, Perhonen MA, Zhang R, Zuckerman JH, Levine BD. Hemodynamics of orthostatic intolerance: implications for gender differences. Am J Physiol Heart Circ Physiol 286: H449–H457, 2004. doi: 10.1152/ajpheart.00735.2002. [PubMed] [CrossRef] [Google Scholar]

23. Halliwill JR. Mechanisms and clinical implications of post-exercise hypotension in humans. Exerc Sport Sci Rev 29: 65–70, 2001. doi: 10.1097/00003677-200104000-00005. [PubMed] [CrossRef] [Google Scholar]

24. Halliwill JR, Buck TM, Lacewell AN, Romero SA. Postexercise hypotension and sustained postexercise vasodilatation: what happens after we exercise? Exp Physiol 98: 7–18, 2013. doi: 10.1113/expphysiol.2011.058065. [PubMed] [CrossRef] [Google Scholar]

25. Halliwill JR, Dinenno FA, Dietz NM. Alpha-adrenergic vascular responsiveness during postexercise hypotension in humans. J Physiol 550: 279–286, 2003. doi: 10.1113/jphysiol.2003.042838. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

26. Halliwill JR, Minson CT, Joyner MJ. Effect of systemic nitric oxide synthase inhibition on postexercise hypotension in humans. J Appl Physiol (1985) 89: 1830–1836, 2000. [PubMed] [Google Scholar]

27. Halliwill JR, Sieck DC, Romero SA, Buck TM, Ely MR. Blood pressure regulation X: what happens when the muscle pump is lost? Post-exercise hypotension and syncope. Eur J Appl Physiol 114: 561–578, 2014. doi: 10.1007/s00421-013-2761-1. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

28. Halliwill JR, Taylor JA, Eckberg DL. Impaired sympathetic vascular regulation in humans after acute dynamic exercise. J Physiol 495: 279–288, 1996. doi: 10.1113/jphysiol.1996.sp021592. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

29. Halliwill JR, Taylor JA, Hartwig TD, Eckberg DL. Augmented baroreflex heart rate gain after moderate-intensity, dynamic exercise. Am J Physiol Regul Integr Comp Physiol 270: R420–R426, 1996. [PubMed] [Google Scholar]

30. Hara K, Floras JS. Influence of naloxone on muscle sympathetic nerve activity, systemic and calf haemodynamics and ambulatory blood pressure after exercise in mild essential hypertension. J Hypertens 13: 447–461, 1995. doi: 10.1097/00004872-199504000-00011. [PubMed] [CrossRef] [Google Scholar]

31. Hayes PM, Lucas JC, Shi X. Importance of post-exercise hypotension in plasma volume restoration. Acta Physiol Scand 169: 115–124, 2000. doi: 10.1046/j.1365-201x.2000.00728.x. [PubMed] [CrossRef] [Google Scholar]

32. Hayward CS, Kelly RP, Collins P. The roles of gender, the menopause and hormone replacement on cardiovascular function. Cardiovasc Res 46: 28–49, 2000. doi: 10.1016/S0008-6363(00)00005-5. [PubMed] [CrossRef] [Google Scholar]

33. Heffernan KS, Collier SR, Kelly EE, Jae SY, Fernhall B. Arterial stiffness and baroreflex sensitivity following bouts of aerobic and resistance exercise. Int J Sports Med 28: 197–203, 2007. doi: 10.1055/s-2006-924290. [PubMed] [CrossRef] [Google Scholar]

34. Heffernan KS, Kelly EE, Collier SR, Fernhall B. Cardiac autonomic modulation during recovery from acute endurance versus resistance exercise. Eur J Cardiovasc Prev Rehabil 13: 80–86, 2006. doi: 10.1097/00149831-200602000-00012. [PubMed] [CrossRef] [Google Scholar]

35. Hill L. Arterial pressure in man while sleeping, resting, working and bathing. J Physiol 22: xxvi–xxx, 1898. [Google Scholar]

36. Jouffroy R, Caille V, Perrot S, Vieillard-Baron A, Dubourg O, Mansencal N. Changes of cardiac function during ultradistance trail running. Am J Cardiol 116: 1284–1289, 2015. doi: 10.1016/j.amjcard.2015.07.045. [PubMed] [CrossRef] [Google Scholar]

37. Kasikcioglu E, Arslan A, Topcu B, Sayli O, Akhan H, Oflaz H, Akin A, Kayserilioglu A, Meric M. Cardiac fatigue and oxygen kinetics after prolonged exercise. Int J Cardiol 108: 286–288, 2006. doi: 10.1016/j.ijcard.2005.03.014. [PubMed] [CrossRef] [Google Scholar]

38. Kenney MJ, Seals DR. Postexercise hypotension. Key features, mechanisms, and clinical significance. Hypertension 22: 653–664, 1993. doi: 10.1161/01.HYP.22.5.653. [PubMed] [CrossRef] [Google Scholar]

39. Kingsley JD, Figueroa A. Acute and training effects of resistance exercise on heart rate variability. Clin Physiol Funct Imaging 36: 179–187, 2016. doi: 10.1111/cpf.12223. [PubMed] [CrossRef] [Google Scholar]

40. Lacewell AN, Buck TM, Romero SA, Halliwill JR. Postexercise syncope: Wingate syncope test and effective countermeasure. Exp Physiol 99: 172–186, 2014. doi: 10.1113/expphysiol.2013.075333. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

41. Lockwood JM, Pricher MP, Wilkins BW, Holowatz LA, Halliwill JR. Postexercise hypotension is not explained by a prostaglandin-dependent peripheral vasodilation. J Appl Physiol (1985) 98: 447–453, 2005. doi: 10.1152/japplphysiol.00787.2004. [PubMed] [CrossRef] [Google Scholar]

42. Lockwood JM, Wilkins BW, Halliwill JR. H1 receptor-mediated vasodilatation contributes to postexercise hypotension. J Physiol 563: 633–642, 2005. doi: 10.1113/jphysiol.2004.080325. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

43. Lucía A, Serratosa L, Saborido A, Pardo J, Boraita A, Morán M, Bandrés F, Megías A, Chicharro JL. Short-term effects of marathon running: no evidence of cardiac dysfunction. Med Sci Sports Exerc 31: 1414–1421, 1999. doi: 10.1097/00005768-199910000-00009. [PubMed] [CrossRef] [Google Scholar]

44. Luttrell MJ, Halliwill JR. Recovery from exercise: vulnerable state, window of opportunity, or crystal ball? Front Physiol 6: 204, 2015. doi: 10.3389/fphys.2015.00204. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

45. Luttrell MJ, Halliwill JR. The intriguing role of histamine in exercise responses. Exerc Sport Sci Rev 45: 16–23, 2017. doi: 10.1249/JES.0000000000000093. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

46. Lynn BM, Minson CT, Halliwill JR. Fluid replacement and heat stress during exercise alter post-exercise cardiac haemodynamics in endurance exercise-trained men. J Physiol 587: 3605–3617, 2009. doi: 10.1113/jphysiol.2009.171199. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

47. MacDonald JR. Potential causes, mechanisms, and implications of post exercise hypotension. J Hum Hypertens 16: 225–236, 2002. doi: 10.1038/sj.jhh.1001377. [PubMed] [CrossRef] [Google Scholar]

48. MacDonald JR, MacDougall JD, Interisano SA, Smith KM, McCartney N, Moroz JS, Younglai EV, Tarnopolsky MA. Hypotension following mild bouts of resistance exercise and submaximal dynamic exercise. Eur J Appl Physiol Occup Physiol 79: 148–154, 1999. doi: 10.1007/s004210050488. [PubMed] [CrossRef] [Google Scholar]

49. MacDougall JD, Tuxen D, Sale DG, Moroz JR, Sutton JR. Arterial blood pressure response to heavy resistance exercise. J Appl Physiol (1985) 58: 785–790, 1985. [PubMed] [Google Scholar]

50. McCord JL, Beasley JM, Halliwill JR. H2-receptor-mediated vasodilation contributes to postexercise hypotension. J Appl Physiol (1985) 100: 67–75, 2006. doi: 10.1152/japplphysiol.00959.2005. [PubMed] [CrossRef] [Google Scholar]

51. McCord JL, Halliwill JR. H1 and H2 receptors mediate postexercise hyperemia in sedentary and endurance exercise-trained men and women. J Appl Physiol (1985) 101: 1693–1701, 2006. doi: 10.1152/japplphysiol.00441.2006. [PubMed] [CrossRef] [Google Scholar]

52. McCord JL, Pellinger TK, Lynn BM, Halliwill JR. Potential benefit from an H1-receptor antagonist on postexercise syncope in the heat. Med Sci Sports Exerc 40: 1953–1961, 2008. doi: 10.1249/MSS.0b013e31817f1970. [PubMed] [CrossRef] [Google Scholar]

53. Meeusen R, Duclos M, Foster C, Fry A, Gleeson M, Nieman D, Raglin J, Rietjens G, Steinacker J, Urhausen A; European College of Sport Science; American College of Sports Medicine . Prevention, diagnosis, and treatment of the overtraining syndrome: joint consensus statement of the European College of Sport Science and the American College of Sports Medicine. Med Sci Sports Exerc 45: 186–205, 2013. doi: 10.1249/MSS.0b013e318279a10a. [PubMed] [CrossRef] [Google Scholar]

54. Middleton N, Shave R, George K, Whyte G, Forster J, Oxborough D, Gaze D, Collinson P. Novel application of flow propagation velocity and ischaemia-modified albumin in analysis of postexercise cardiac function in man. Exp Physiol 91: 511–519, 2006. doi: 10.1113/expphysiol.2005.032631. [PubMed] [CrossRef] [Google Scholar]

55. Montiel G, Horn T, Vafa R, Solera A, Hollmann W, Predel HG, Brinkmann C. Echocardiographic and biochemical analysis of cardiac function and injury among female amateur runners post-marathon. Wien Klin Wochenschr 128: 193–197, 2016. doi: 10.1007/s00508-015-0881-9. [PubMed] [CrossRef] [Google Scholar]

56. Moralez G, Romero SA, Rickards CA, Ryan KL, Convertino VA, Cooke WH. Effects of dehydration on cerebrovascular control during standing after heavy resistance exercise. J Appl Physiol (1985) 112: 1875–1883, 2012. doi: 10.1152/japplphysiol.01217.2011. [PubMed] [CrossRef] [Google Scholar]

57. Neilan TG, Yoerger DM, Douglas PS, Marshall JE, Halpern EF, Lawlor D, Picard MH, Wood MJ. Persistent and reversible cardiac dysfunction among amateur marathon runners. Eur Heart J 27: 1079–1084, 2006. doi: 10.1093/eurheartj/ehi813. [PubMed] [CrossRef] [Google Scholar]

58. Niemelä TH, Kiviniemi AM, Hautala AJ, Salmi JA, Linnamo V, Tulppo MP. Recovery pattern of baroreflex sensitivity after exercise. Med Sci Sports Exerc 40: 864–870, 2008. doi: 10.1249/MSS.0b013e3181666f08. [PubMed] [CrossRef] [Google Scholar]

59. Niijima-Yaoita F, Tsuchiya M, Ohtsu H, Yanai K, Sugawara S, Endo Y, Tadano T. Roles of histamine in exercise-induced fatigue: favouring endurance and protecting against exhaustion. Biol Pharm Bull 35: 91–97, 2012. doi: 10.1248/bpb.35.91. [PubMed] [CrossRef] [Google Scholar]

60. Peake JM, Markworth JF, Nosaka K, Raastad T, Wadley GD, Coffey VG. Modulating exercise-induced hormesis: does less equal more? J Appl Physiol (1985) 119: 172–189, 2015. doi: 10.1152/japplphysiol.01055.2014. [PubMed] [CrossRef] [Google Scholar]

61. Peçanha T, Bartels R, Brito LC, Paula-Ribeiro M, Oliveira RS, Goldberger JJ. Methods of assessment of the post-exercise cardiac autonomic recovery: a methodological review. Int J Cardiol 227: 795–802, 2017. doi: 10.1016/j.ijcard.2016.10.057. [PubMed] [CrossRef] [Google Scholar]

62. Pellinger TK, Dumke BR, Halliwill JR. Effect of H1- and H2-histamine receptor blockade on postexercise insulin sensitivity. Physiol Rep 1: e00033, 2013. doi: 10.1002/phy2.33. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

63. Pellinger TK, Simmons GH, Maclean DA, Halliwill JR. Local histamine H(1-) and H(2)-receptor blockade reduces postexercise skeletal muscle interstitial glucose concentrations in humans. Appl Physiol Nutr Metab 35: 617–626, 2010. doi: 10.1139/H10-055. [PubMed] [CrossRef] [Google Scholar]

64. Piepoli M, Coats AJ, Adamopoulos S, Bernardi L, Feng YH, Conway J, Sleight P. Persistent peripheral vasodilation and sympathetic activity in hypotension after maximal exercise. J Appl Physiol (1985) 75: 1807–1814, 1993. [PubMed] [Google Scholar]

65. Pricher MP, Holowatz LA, Williams JT, Lockwood JM, Halliwill JR. Regional hemodynamics during postexercise hypotension. I. Splanchnic and renal circulations. J Appl Physiol (1985) 97: 2065–2070, 2004. doi: 10.1152/japplphysiol.00465.2004. [PubMed] [CrossRef] [Google Scholar]

66. Privett SE, George KP, Whyte GP, Cable NT. The effectiveness of compression garments and lower limb exercise on post-exercise blood pressure regulation in orthostatically intolerant athletes. Clin J Sport Med 20: 362–367, 2010. doi: 10.1097/JSM.0b013e3181f20292. [PubMed] [CrossRef] [Google Scholar]

67. Queiroz AC, Rezk CC, Teixeira L, Tinucci T, Mion D, Forjaz CL. Gender influence on post-resistance exercise hypotension and hemodynamics. Int J Sports Med 34: 939–944, 2013. doi: 10.1055/s-0033-1337948. [PubMed] [CrossRef] [Google Scholar]

67a. Rezk CC, Marrache RC, Tinucci T, Mion D Jr, Forjaz CL. Post-resistance exercise hypotension, hemodynamics, and heart rate variability: influence of exercise intensity. Eur J Appl Physiol 98: 105–112, 2006. doi: 10.1007/s00421-006-0257-y. [PubMed] [CrossRef] [Google Scholar]

68. Roberts LA, Muthalib M, Stanley J, Lichtwark G, Nosaka K, Coombes JS, Peake JM. Effects of cold water immersion and active recovery on hemodynamics and recovery of muscle strength following resistance exercise. Am J Physiol Regul Integr Comp Physiol 309: R389–R398, 2015. doi: 10.1152/ajpregu.00151.2015. [PubMed] [CrossRef] [Google Scholar]

69. Roberts LA, Nosaka K, Coombes JS, Peake JM. Cold water immersion enhances recovery of submaximal muscle function after resistance exercise. Am J Physiol Regul Integr Comp Physiol 307: R998–R1008, 2014. doi: 10.1152/ajpregu.00180.2014. [PubMed] [CrossRef] [Google Scholar]

70. Roberts LA, Raastad T, Markworth JF, Figueiredo VC, Egner IM, Shield A, Cameron-Smith D, Coombes JS, Peake JM. Post-exercise cold water immersion attenuates acute anabolic signalling and long-term adaptations in muscle to strength training. J Physiol 593: 4285–4301, 2015. doi: 10.1113/JP270570. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

71. Romero SA, Cooke WH. Hyperventilation before resistance exercise: cerebral hemodynamics and orthostasis. Med Sci Sports Exerc 39: 1302–1307, 2007. doi: 10.1249/mss.0b013e3180653636. [PubMed] [CrossRef] [Google Scholar]

72. Romero SA, Ely MR, Sieck DC, Luttrell MJ, Buck TM, Kono JM, Branscum AJ, Halliwill JR. Effect of antioxidants on histamine receptor activation and sustained postexercise vasodilatation in humans. Exp Physiol 100: 435–449, 2015. doi: 10.1113/EP085030. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

73. Romero SA, Hocker AD, Mangum JE, Luttrell MJ, Turnbull DW, Struck AJ, Ely MR, Sieck DC, Dreyer HC, Halliwill JR. Evidence of a broad histamine footprint on the human exercise transcriptome. J Physiol 594: 5009–5023, 2016. doi: 10.1113/JP272177. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

74. Romero SA, McCord JL, Ely MR, Sieck DC, Buck TM, Luttrell MJ, MacLean DA, Halliwill JR. Mast cell degranulation and de novo histamine formation contribute to sustained post-exercise vasodilation in humans. J Appl Physiol (1985) 122: 603–610, 2016. doi: 10.1152/japplphysiol.00633.2016. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

75. Seiler S, Haugen O, Kuffel E. Autonomic recovery after exercise in trained athletes: intensity and duration effects. Med Sci Sports Exerc 39: 1366–1373, 2007. doi: 10.1249/mss.0b013e318060f17d. [PubMed] [CrossRef] [Google Scholar]

76. Sieck DC, Ely MR, Romero SA, Luttrell MJ, Abdala PM, Halliwill JR. Post-exercise syncope: Wingate syncope test and visual-cognitive function. Physiol Rep 4: e12883, 2016. doi: 10.14814/phy2.12883. [PMC free article] [PubMed] [CrossRef] [Google Scholar]

77. Teixeira L, Ritti-Dias RM, Tinucci T, Mion Júnior D, Forjaz CL. Post-concurrent exercise hemodynamics and cardiac autonomic modulation. Eur J Appl Physiol 111: 2069–2078, 2011. doi: 10.1007/s00421-010-1811-1. [PubMed] [CrossRef] [Google Scholar]

78. Thijs RD, Reijntjes RH, van Dijk JG. Water drinking as a potential treatment for idiopathic exercise-related syncope: a case report. Clin Auton Res 13: 103–105, 2003. doi: 10.1007/s10286-003-0083-9. [PubMed] [CrossRef] [Google Scholar]

79. Wieling W, van Dijk N, Thijs RD, de Lange FJ, Krediet CT, Halliwill JR. Physical countermeasures to increase orthostatic tolerance. J Intern Med 277: 69–82, 2015. doi: 10.1111/joim.12249. [PubMed] [CrossRef] [Google Scholar]

80. Wilkins BW, Minson CT, Halliwill JR. Regional hemodynamics during postexercise hypotension. II. Cutaneous circulation. J Appl Physiol (1985) 97: 2071–2076, 2004. doi: 10.1152/japplphysiol.00466.2004. [PubMed] [CrossRef] [Google Scholar]

81. Willie CK, Ainslie PN, Taylor CE, Eves ND, Tzeng Y-C. Maintained cerebrovascular function during post-exercise hypotension. Eur J Appl Physiol 113: 1597–1604, 2013. doi: 10.1007/s00421-012-2578-3. [PubMed] [CrossRef] [Google Scholar]

82. Wilson TE, Cui J, Zhang R, Witkowski S, Crandall CG. Skin cooling maintains cerebral blood flow velocity and orthostatic tolerance during tilting in heated humans. J Appl Physiol (1985) 93: 85–91, 2002. doi: 10.1152/japplphysiol.01043.2001. [PubMed] [CrossRef] [Google Scholar]

83. Yoneda H, Niijima-Yaoita F, Tsuchiya M, Kumamoto H, Watanbe M, Ohtsu H, Yanai K, Tadano T, Sasaki K, Sugawara S, Endo Y. Roles played by histamine in strenuous or prolonged masseter muscle activity in mice. Clin Exp Pharmacol Physiol 40: 848–855, 2013. doi: 10.1111/1440-1681.12167. [PubMed] [CrossRef] [Google Scholar]

84. Thompson PD. A knockout finish: advice on preventing a finish-line fainting spell. Runner’s World 2004. [Google Scholar]


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Hemodynamic adjustments during the recovery from aerobic and resistance exercise

Hemodynamic ParameterPostaerobicPostresistance
Mean arterial pressure
Cardiac output
Systemic vascular conductance↑↑
Skeletal muscle conductance↑↑?
Splanchnic and renal conductances?
Cutaneous conductance?
Cerebral conductance?